MRI scans show that running may affect the structure and function of the brain in ways similar to complex tasks like playing a musical instrument
Researchers at the University at Buffalo Research Institute on Addictions have discovered how a gene in the brain’s dopamine system can play an important role in prolonging lifespan: it must be coupled with a healthy environment that includes exercise.
The study, led by Panayotis (Peter) K. Thanos, senior research scientist at RIA, appears in the current, online version of Oncotarget Aging.
Researchers from the David Geffen School of Medicine at UCLA found that cardiovascular disease patients who have high muscle mass and low fat mass have a lower mortality risk than those with other body compositions. The findings also suggest that regardless of a person’s level of fat mass, a higher level of muscle mass helps reduce the risk of death.
This findings indicate the importance of assessing body composition as a way to help predict cardiovascular and total mortality in people with cardiovascular disease.
Japanese researchers have taken a step closer to creating electronic skin—e-skin—by employing flexible electronics that can be worn as a second skin for biomedical and other applications. The aim is to have e-skin eventually become as much a part of our daily lives as the clothes we wear.
If this eventuates, athletes will use e-skin to view their heart rates, sugar levels and work rate. It could provide doctors with continuous data on patients’ vital signs without the need for repeatedly attaching and removing medical equipment, while the rest of us might employ it to monitor our body health metrics. Meanwhile, engineers could put down their tablets when doing tricky repair work, and instead view maintenance procedures displayed on their arms.
Could an unhealthy diet and lack of exercise be making you age faster? Researchers at Mayo Clinic believe there is a link between these modifiable lifestyle factors and the biological processes of aging. In a recent study, researchers demonstrated that a poor diet and lack of exercise accelerated the onset of cellular senescence and, in turn, age-related conditions in mice. Results appear today inDiabetes.
Senescent cells are cells that contribute to diseases and conditions associated with age. Researchers from the Mayo Clinic Robert and Arlene Kogod Center on Aging found that exercise prevents premature senescent cell accumulation and protects against the damaging effects of an unhealthy diet, including deficiencies in physical, heart, and metabolic function, equivalent to diabetes.